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Rheumatic Fever and Heart Disease* By Dr. Royal Lee

The relationship between rheumatic fever and heart disease has always been a mystery. Doctors Aaron Keilner and Theodore Robertson of New York announced their discovery ofa "missing link" in this mystery at the Second World Congress of Cardiology and the 27th Scientific Session of American Heart Association at Washington, November, 1954.
This discovery was the demonstration that a proteolytic enzyme ftom the streptococcus had the ability to cause specific damage to heart valves and muscle when injected into test animals. They called the enzyme a "potent cardiotoxin."
Those who have given a little thought to Applied Protomorphology need no more explanation as to how this streptococcus enzyme works. It releases into the blood stream the protein residues ftom heart tissue which then act as "foreign proteins" and instigate the formation of antibodies to these heart proteins (Natural Tissue Antibody!). These antibodies circulate in the blood, and actually inhibit the repair processes of the heart, according to the clinical evidence available by observing any rheumatic fever patient.
If we supply an antigenic extract of heart tissue (beef heart - PMG) for oral administration, it is absorbed through the mucous membrane of the alimentary tract, meets the heart antibody in the blood stream, and REDUCES THIS INHIBITORY EFFECT so promptly that if a cardiographic recording is made before and 15 minutes after ingestion of CARDIOTROPHIN PMG (beef heart extract), very pronounced improvement in the heart function is observable in the victim of excess cardiac antibody.
This is one of the measurable reactions that cannot be attributed to psychosomatic suggestion by the hypercritical skeptic of progress. The ENDOCARDIOGRAPH (phonocardiograph) is an ideal device to demonstrate this effect. Patients who have had a coronary lesion also commonly have triggered off the production of cardiac antibodies and appear to be suffering ftom a debilitated state of heart tissue which heals far more slowly than normal.
In our discussion of the sedimentation rate, we noted that an increased sedimentation rate was common in rheumatic fever, and that it resulted ftom the release into the blood stream of tissue residues that were abnormal. It is obvious that an enzyme like this streptococcal cardiotoxin would be active in creating a high sedimentation rate.
Our normal defense to such potent bacterial invasions is an intact adrenal, secondary to good nutrition. Refined and cooked foods, lacking the vitamins required by the adrenal (the C Complex, the B Complex with special reference to the tyrosinase in the first and the pantothenic acid in the second) set the stage for an attack of rheumatic fever. Rinehart has reported the value of vitamin P in reducing the sedimentation rate in rheumatic fever. The relationship of vitamin E Complex to heart disease becomes apparent here. In vitamin E deficiency the cell chromosomes disintegrate. This releases the antigenic tissue factors that create the natural tissue antibody which thereafter inhibits heart repair. The muscle cell damage known to follow vitamin B deficiency is, we feel, directly due to this process. The most critically active function of vitamin E is to prevent oxidative destruction of the protective insulating wrapping of the determinant "blue-prints" of the cell chromosome. The oxygen demand of the tissues may rise to 250% of normal by reason of vitamin E deficiency and this effect, it is very obvious, immediately throws a tremendous unnecessary load on the heart, as the blood volume to be pumped is directly proportional to oxygen demand. The B Complex deficiency creates a terrific overload and at the very same time undermines the integrity of the muscular tissue by promoting the production of heart tissue antibody.

No wonder test animals placed on a diet of no vitamin B but everything else necessary soon drop dead from heart failure-at the rate of about 50% per year.
The vitamin C Complex with its vitamin P component is synergistic in that ascorbic acid helps the antioxidant reaction of the B, while the P factor provides calcium in the proper fOnD to aid thrombin fonnation, the precursor to connective tissue, so that capillary weakness is forestalled, and the protein intercellular substance is maintained. Scurvy is inherently connective tissue disintegration.
The C Complex has an additional physiological function that of promoting increased oxygen-carrying capacity of erythrocytes, helping thereby to REDUCE the load on the heart. With so many factors cooperating to maintain physiological balances, it is easy to comprehend the sudden death from a combined effect of the multiple forms of vitamin deficiency.

Vitamin F, the unsaturated fatty acid complex, now newly in the news since its relationship to cholesterol control is established is important to the heart, since the second heart sound disappears in case of deficiency. Why is this? Simply that vitamin F promotes calcium diffusion from the blood to muscle cell, and where the heart muscle is starved of this element the contraction cycle collapses before completion of the contraction, resulting in a tapering off of velocity of outgoing blood from the heart, so that there is no check-valve sound ftom aortic or pulmonary back-pressure. Sometimes one area shows the missing sound, sometimes another, depending on the state of the right or left ventricular muscle. If pulmonary hypertension ftom weak adrenals is present, the right side will be the most fatigued, and show the most definite weakening or loss of the second sound.
Vitamin F is a member of the vitamin E complex; it was first discovered in wheat genu oil. It is not present in any wheat or grain product unless it has been milled within a few days of its conversion into bread or other food. It is lost by oxidation, like the tocopherol group, at the rate of ten percent a day in flour or meal, after the wax seal of the intact seed has been broken Heat does not affect these vitamins, but like vitamin K, the oxygen of the air makes quick work of them once their natural protective mechanism is damaged.
We might point out here the newly found fact that vitamin K, lost in vegetable foods stored in a deep freezer, protects us against coronary thrombosis, and there is a 3000 percent increase in coronary thrombosis in areas where there has occurred a general use of deep fteeze units.
We seem to fmd it hard to circumvent basic laws of nutrition. Nature has capital punishment waiting for the law violator, but it is pathetic to realize no warnings are available for the innocent and gullible victims of civilization. As far back as 1911 tooth decay was known to be a disease only contracted by eaters of the refined foods of civilization but few people are told that the food we eat today is the body we live in tomorrow, and that if we fail to eat tissue-building foods our body will progressively disintegrate and decayed teeth are a warning of more trouble to come; arthritis, heart disease, liver disease, and even mental disease. (MENTAL DISEASE may even precede all other symptoms of pellagra (vitamin G complex deficiency) says Harris in his book "Clinical Pellagra", and an actual psychosis of a severe type may be the first manifestation).
Heart disease and mental symptoms, however, are a lot easier to treat by nutritional replacement than arthritis and dental disease where structural damage precedes the symptoms; it is a lot harder for Nature to rebuild destroyed tissues than it is for her to correct functional disorders. Soft tissues regenerate a lot more successfully than bones and teeth.
"The studies described suggest that the nonnal body does indeed possess means for defending itself against cancer and that these means are wanting in patients with advanced disease." (SloanKettering Institute for Cancer Research Report, 1957, p.3). Compare this with the fact that cancer cells implanted into test animals fail to grow unless they have been fed refined foods for some time previous. This looks like the simple matter of addition; 2 plus 2 equals 4.

* "By far the greatest number of valvular lesions results from rheumatic fever. Rheumatic fever is an autoimmune or allergic disease in which the heart valves are likely to be damaged or destroyed. It is initiated by streptococcal toxin in the following manner..." Textbook a/Medical Physiology, Arthur Guyton MD. 1" Edition. Saunders. Page 318.

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